All rodents injected were dead within days say scientists.
Creating killer diseases in a lab to prepare a 'defence' against them if they ever appeared in nature is the preferred story of Covid-19. Now, as the WEF talk endlessly about Disease-X, Scientists from China claim that they have purposefully created a virus that has a '100%' kill rate in infected mice.
say officials. Critics of the study said: 'This madness must be stopped before it's too late'
Scientists in Beijing, linked to the Chinese military, claim they have cloned a Covid-like virus found in pangolins, known as GX_P2V, and used it to infect mice. The mice had been 'humanized', meaning they were engineered to express a protein found in humans with the goal being to assess how the virus might react in humans. The result was that all eight rodents were dead in eight days, which 'researchers' appeared to be revelling in.
Every rodent that was infected with the pathogen died within eight days, which the researchers described as 'surprisingly' quick.
The mailonline.com reports:
Professor Francois Balloux, an infectious disease expert based at University College London, wrote on Twitter (X): 'It's a terrible study, scientifically totally pointless.
'"I can see nothing of vague interest that could be learned from force-infecting a weird breed of humanized mice with a random virus. Conversely, I could see how such stuff might go wrong...'"
Professor Richard Ebright, a chemist at Rutgers University in New Brunswick, New Jersey, told DailyMail.com he wholeheartedly agreed with Professor Balloux's assessment.
He added: 'The preprint does not specify the biosafety level and biosafety precautions used for the research.
'The absence of this information raises the concerning possibility that part or all of this research, like the research in Wuhan in 2016-2019 that likely caused the Covid-19 pandemic, recklessly was performed without the minimal biosafety containment and practices essential for research with a potential pandemic pathogens.'
According to the study, carried out by the Beijing University of Chemical Technology, the virus was discovered in 2017 prior to the Covid outbreak. It was discovered in Malaysia in pangolins - scaly mammals that are known harborers of coronaviruses and were heavily speculated to be the intermediate host that passed Covid from bats to humans.
The researchers cloned the virus and stored multiple copes in the Beijing lab, where it continued to evolve. It is unclear when the newly surfaced study was conducted. But the researchers said it was possible the virus had undergone a 'virulence-enhancing mutation' in storage, which made it more deadly.
For the new research, eight mice were infected with the virus, eight were infected with an inactivated virus and eight were used as a control group. All mice infected with the virus died. They succumbed to the infection between seven and eight days after being infected.
Symptoms included their eyes turning completely white, rapid weight loss and fatigue.
Researchers found 'significant amounts' of the virus in the rodents' brains, lungs, noses, eyes and windpipes.
By day six, the viral load had 'significantly decreased' in the lungs, but the animals' brains had shrunk and there were 'exceptionally high' virus levels in their brains. The results suggest that the virus infects via the respiratory system and then migrates to the brain - unlike Covid which causes lower lung infections and pneumonia in severe cases. However, there have been examples of Covid being found in brain tissue of severely sick patients.
'Severe brain infection during the later stages of infection may be the key cause of death in these mice,' the researchers said. They concluded: 'This is the first report showing that a SARS-CoV-2-related pangolin coronavirus can cause 100 percent mortality in hACE2 mice, suggesting a risk for GX_P2V to spill over into humans.'
However, the original strain of Covid also killed 100 percent in mice in some studies, meaning the new results may not be directly applicable to humans. Dr Gennadi Glinsky, a retired professor of medicine at Stanford, said on social media: 'This madness must be stopped before [it is] too late.'
This is either incredibly stupid, or the WHO are softening us up for Disease 'X'.